All terms in HP

Label Id Description
regulation of inflammatory response to wounding GO_0106014 [Any process that modulates the frequency, rate or extent of the inflammatory response to wounding.]
positive regulation of inflammatory response to wounding GO_0106016 [Any process that activates or increases the frequency, rate or extent of the inflammatory response to wounding.]
negative regulation of inflammatory response to wounding GO_0106015 [Any process that stops, prevents, or reduces the frequency, rate or extent of the inflammatory response to wounding.]
Glomerular hyalinosis away from the vascular and tubular poles HP_0033219 [Accumulation of amorphous, eosinophilic, glassy, periodic acid-Schiff (PAS)-positive, silver-negative material in the glomerulus at neither tip nor perihilar/vascular poles. Both the vascular and the tubular pole are present in the glomerular cross section.]
Glomerular hyalinosis HP_0033216 [Accumulation of amorphous, eosinophilic, glassy, periodic acid-Schiff (PAS)-positive, silver-negative material in the glomerulus. Distribution can include vascular pole, perihilar, tip or neither tip, perihilar or vascular.]
Glomerular hyalinosis at the tubular pole HP_0033218 [Accumulation of amorphous, eosinophilic, glassy, periodic acid-Schiff (PAS)-positive, silver-negative material in the glomerulus at the tip/tubular pole.]
Perihilar glomerular hyalinosis HP_0033217 [Accumulation of amorphous, eosinophilic, glassy, periodic acid-Schiff (PAS)-positive, silver-negative material in the glomerulus at the vascular pole/perihilum.]
Obliterative abnormality of the renal glomerulus HP_0033215
Adrenocorticotropin receptor defect HP_0008259 [Adrenal insufficiency secondary to a defect in the ACTH receptor.]
Congenital adrenal hyperplasia HP_0008258 [A type of adrenal hyperplasia with congenital onset.]
Adrenal hyperplasia HP_0008221 [Enlargement of the adrenal gland.]
Transient neonatal diabetes mellitus HP_0008255
Abnormality of the pancreatic islet cells HP_0006476 [An abnormality of the islet of Langerhans, i.e., of the regions of the pancreas that contain its endocrine cells. These are the alpha cells, which produce glucagon, the beta cells, which produce insulin and amylin, the delta cells, which produce somatostatin, the PP cells, which produce pancreatic polypeptide, and the epsilon cells, which produce ghrelin.]
Recurrent viral pneumonia HP_0033214 [Increased susceptibility to viral pneumonia, as manifested by recurrent episodes of viral pneumonias.]
Recurrent pneumonia HP_0006532 [An increased susceptibility to pneumonia as manifested by a history of recurrent episodes of pneumonia.]
Elevated urine suberic acid level HP_0033213 [The concentration of suberic acid in the urine, normalized for urine concentration, is above the upper limit of normal.]
Decreased total iron binding capacity HP_0033211 [A reduction in the total-iron binding capacity, which measures how much serum iron is bound if an excess of radioactive iron is added. The latent (or free) iron binding capacity is the difference between the TIBC and the measured serum iron, corresponding to the transferrin not bound to iron, i.e., free iron binding capacity.]
Congenital alveolar dysplasia HP_0033210 [Arrest of lung development in the cananicular stage (weeks 18 to 26 of human gestation) resulting in simplified acinar spaces, frequently with abundant intervening mesenchyme and no alveoli. In later arrest growth stages early saccular formations may be seen. May resemble the lobular maldevelopment often seen in alveolar capillary dysplasia/misaligment of the pulmonary veins without vein misalignment or marked hypertensive changes of the pulmonary arteries.]
Acinar dysplasia HP_0033209 [Arrest of lung development in the pseudoglandular stage (weeks 8 to 16 of human gestation) resulting in small sized lungs, predominantly composed of bronchial and bronchiolar structures embedded in abundant loose mesenchyme that is poorly vascularized. Acinar structures are essentially absent, with no significant formation of saccules and no alveoli.]
Alveolar capillary dysplasia HP_0033208 [A developmental defect of the alveolar airspace during cananicular lung development characterized by a decreased number of pulmonary capillaries located away from the alveolar epithelium, thickened alveolar septae, media hypertrophy of small pulmonary arteries and muscularization of distal arterioles, and in some cases lymphangiectasis.]